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Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. However, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK signifies elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by greater pFAK localization in 5-HT synapses. Our data https://pro33-login90099.targetblogs.com/32827746/pro33-login-can-be-fun-for-anyone

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